Montoya
acjzi****@jmora*****
2007年 6月 17日 (日) 20:01:55 JST
This illustrates the large amount of research being conducted in this area, and constantly emerging knowledge that results. And we might develop new enzymes which could break down those substances that the body, unaided, cannot deal with. Now that Benowitz has isolated oncomodulin, he believes even greater regeneration is possible by combining it with agents that counteract growth inhibitors. The changes, recommended late last year by a government-commissioned independent review, would permit therapeutic cloning under strict controls. white blood cells from that mouse's descendants were injected into ordinary mice with cancer and their disease was completely wiped out. Normal brain tissue does not have telomerase, so this virus replicates only in cancer cells. Comparatively little at this point, sad to say. I think that many of the readers of this newsletter will find it a useful improvement for research both personal and professional; see this Fight Aging! Exercise is the most beneficial strategy to treat atrophy. Here is another: research "shows that oxygen free radicals are damaging proteins in mitochondria, the tiny cellular 'batteries' of brain cells. The need for such technologies is great. This thus led to the 'telomere hypothesis of dementia,' that asks whether telomere shortening contributes to the genesis of certain age-related diseases, such as dementia . This accumulation is one root cause of aging, and so the more we know about DNA repair, the better. " A point of contention in the gerontology community is that scientists already know enough to be working towards meaningful anti-aging medicine - not just gathering more data. " File this under "how good health practices actually work. " Given the level of scientific backing for the present consensus view of evolution and aging, Mitteldorf has an uphill road ahead. XMLVia Reuters, a good illustration of the interconnected nature of the body. A number of research groups have published recently on the relationship between age-related mitochondrial DNA damage and Parkinson's disease. "I don't believe that if you give most people longer lives, even in better health, they are going to find new opportunities and new initiatives. >From EurekAlert, news of a gene that "likely influences the ability of the pancreas to recruit a type of cell essential for constructing the walls of blood vessels. Here is an update from the Boston Globe: "Fauza's work focuses on the trachea and the diaphragm because the structures needed to fix them are relatively simple. I have decided that I should act in support of healthy life extension research, for reasons both selfish and altruistic. You can find a roundup of the coverage most relevant to healthy life extension over at Fight Aging! This will change the way we have children, change the way we manage our emotional states and will even alter our life spans. barring a terminally ill patient from use of a potentially life-saving treatment impinges on this right of self-preservation. Plugging holes in the failing dam is not a viable path forward to healthy life extension. The 'common' deletion, a marker mitochondrial DNA-deletion for accumulating oxidative stress damage, occurred most prominently in graying hair follicles. " The strongly positive effects on human health have been handily demonstrated in recent years - you owe it to yourself to look into calorie restriction. -------------- next part -------------- HTML$B$NE:IU%U%!%$%k$rJ]4I$7$^$7$?(B...下載 -------------- next part -------------- $B%F%-%9%H7A<00J30$NE:IU%U%!%$%k$rJ]4I$7$^$7$?(B... $B%U%!%$%kL>(B: $BL5$7(B $B7?(B: image/gif $B%5%$%:(B: 4748 $B%P%$%H(B $B ＠ bL@(B: $BL5$7(B URL: http://lists.sourceforge.jp/mailman/archives/honsure-messenger/attachments/20070617/807a0344/attachment.gif
